Dr. Mikovits holds a PhD in Biochemistry and Molecular Biology from George Washington University. Her dissertation was on HIV latency and mechanisms of immune activation in monocytes. Dr. Mikovits was a Postdoctoral Scholar in Molecular Virology at the Laboratory of Genomic Diversity, National Cancer Institute under Dr. David Derse.<\/p>\n Over the past 26 years, she has published 51 scientific papers in peer-reviewed journals, and worked as a government scientist for many years developing viruses and vaccinations. In 2011 when she made a horrifying discovery that was contaminating all vaccinations, she presented her data to government officials and was threatened & told to destroy all her data.<\/p>\n When she was jailed, her career systematically destroyed, and a gag order put in place for 4 years threatening that if she spoke out she would be thrown back in jail.<\/strong>\u00a0That gag order has just lifted, and she\u2019s dumped the government right in it!<\/p>\n She speaks in this interview with Candyce Estave about how autism is associated with vaccines, also cancers, chronic fatigue syndrome, Alzheimers, auto immune diseases, allergies and more.<\/p>\n Notice at minute 6:20 \u201cThe Brain and Immune System are Linked at birth\u201d<\/strong><\/p>\n She discusses how the cocktail of vaccinations pumped into babies mutate to develop months and years down the track into new viruses, cancers and diseases, some they don\u2019t even know about yet.<\/p>\n She further explains how the viruses injected through vaccines tear open our DNA and insert their own DNA to mutate our genetic makeup and be passed on generation after generation.<\/p>\n Here is some of the research she has studied and references in this interview:<\/p>\n 1. \u00a0Antipurinergic Therapy Corrects the Autism-Like Features in the Poly (IC) Mouse Model<\/u>\u00a0\u2013 In early 2013, Dr. Robert Naviaux, a Professor\u00a0<\/strong>of Genetics and co-director of the Mitochondrial and Metabolomic Disease Center at the University of California, San Diego, as well as the recipient of an Autism Speaks\u00a0International Trailblazer<\/i>\u00a0award reported some of the most exciting autism news in years. \u00a0In an article published inScience Daily\u00a0<\/i>on March 13, 2013, Naviaux explained, \u201cWhen cells are exposed to classical forms of danger, such as a virus, infection, or toxic environmental substance, a defense mechanism is activated. \u00a0This results in changes to metabolism and gene expression, and reduces the communication between neighboring cells. \u00a0Simply put, when cells stop talking to each other, children stop talking.\u201d \u00a0In PLOSOne Naviaux and his team reported that administration of an old drug, suramin, (traditionally used to treat the parasite which causes African sleeping sickness) restored cell to cell communication in a mouse model of autism, raising hopes that a similar result might be obtained in\u00a0humans.\u00a0http:\/\/www.sciencedaily.com\/releases\/2013\/03\/130313182019.htm<\/a>\u00a0http:\/\/www.ncbi.nlm.nih.gov\/pubmed\/23516405<\/a><\/strong><\/p>\n 2.\u00a0\u00a0War and Peace between Microbes: HIV-1 Interactions with Coinfecting Viruses<\/u>\u00a0\u2013\u00a0This\u00a0 2009 article from the Journal\u00a0Cell Host and Microbe<\/i>\u00a0explains how a retrovirus can disrupt the equilibrium between a host and its microbes, resulting in a reactivation of persistent viruses, or invasion by new viruses, as well as resulting in either immuno-deficiency or immuno-activation.\u00a0<\/strong><\/p>\n 3. \u00a0<\/strong>Generation of Multiple Replication-Competent Retroviruses through Recombination between PreXMRV-1 and PreXMRV-2<\/u>\u00a0\u2013 This research from Dr. John Coffin and Dr. Vinay Pathak showed that replication competent murine leukemia retroviruses can\u00a0be produced in a relatively short period of time.\u00a0 From their abstract, \u201cTo determine their potential to generate RCRs (author\u2019s note \u2013 replication-competent retroviruses), we transfected PreXMRV-1 and PreXMRV-2 into 293T cells and used the virus produced to infect fresh cells; the presence of reverse transcriptase activity at 10 days indicated the presence of RCRs.\u201d<\/strong>\u00a0http:\/\/www.ncbi.nlm.nih.gov\/pubmed\/23966380<\/a><\/strong><\/p>\n 4. \u00a0Frequent Detection of Infectious Xenotropic Murine Leukemia Virus (XMLV) in Human Cultures Established from Mouse Xenografts<\/u>\u00a0\u2013 This chilling research from the well-regarded Dr. Adi Gazdar, suggests that the murine leukemia retroviruses may have the potential to become airborne.\u00a0 In their conclusion the Gazdar team warned, \u201cLaboratories working with xenograft-derived human cultures should be aware of the risk of contamination with potentially bio-hazardous human-tropic mouse viruses and their spread to other cultures.\u201d\u00a0http:\/\/www.ncbi.nlm.nih.gov\/pmc\/articles\/PMC3218386\/<\/a>\u00a0<\/strong>\u00a0\u00a0<\/strong><\/p>\n 5. \u00a0<\/strong>Xenotropic Murine Leukemia Virus-related Virus-associated Chronic Fatigue Syndrome Reveals a Distinct Inflammatory Signature<\/u>\u00a0\u2013\u00a0(T<\/strong>he cytokine signature paper was published in 2010 and first presented in CFS patients at IiME in London in 2008).\u00a0<\/strong>This research by PLAGUE co-author, Dr. Judy A. Mikovits, was the first to identify a signature of 10 cytokines and chemokines which would correctly identify XMRV\/CFS patients with a 93% specificity and 96% sensitivity.\u00a0http:\/\/www.ncbi.nlm.nih.gov\/pubmed\/21576403<\/a><\/strong><\/p>\n 6. \u00a0<\/strong>Xenotropic MLV Envelope Proteins Induce Tumor Cells to Secrete Factors that Promote the Formation of Immature Blood Vessels<\/u><\/strong>\u00a0\u2013 This 2013 research by Dr. Gary Owens of the University of Virginia provides evidence that envelope proteins on the surface of murine leukemia viruses can affect cancer progression.\u00a0 The article states, \u201cThe studies described herein address these questions, and show that at least one other XMRV-like virus exists, and that the virus evolved the ability to infect human cells and to express gene products that impact tumor pathogenesis.\u201d\u00a0 Dr. Mikovits will never forget how even at the XMRV Conference held at the Cleveland Clinic in November of 2009, Dr. Owens was asserting that more than one type of xenotropic murine leukemia virus might exist, as is the case with other known human retroviruses.\u00a0 Dr. Mikovits will also always remember the kindness with which Dr. Owens invited her to a series of lectures timed to coincide with the 30th anniversary of her graduation from the University of Virginia.\u00a0http:\/\/www.ncbi.nlm.nih.gov\/pubmed\/23537062<\/a><\/strong><\/p>\n \u200b7. \u00a0<\/strong>Neurotoxic Effects of Postnatal Thimerosal are Mouse Strain Dependent<\/u><\/strong>\u00a0<\/strong>\u00a0\u2013 This work from Dr. Ian Lipkin and Dr. Mady Hornig was the first to demonstrate that auto-immune sensitive mice had a decreased ability to handle the hazard posed by mercurials used in common childhood vaccines and developed behaviors reminiscent of autism. \u00a0PLAGUE co-author, Kent Heckenlively would like to express his appreciation for Dr. Ian Lipkin\u2019s kind invitation to attend a cocktail reception on September 19, 2013 at the SoHo Grand Hotel in Manhattan, honoring the work of Columbia University in chronic fatigue syndrome\/ME and autism. \u00a0At the event Kent especially enjoyed a series of delightful conversations with the brilliant, brave, and engaging\u00a0Dr. Mady Hornig about her\u00a0research.\u00a0http:\/\/www.ncbi.nlm.nih.gov\/pubmed\/15184908<\/a><\/strong><\/p>\n 8.\u00a0\u00a0Identification, Viral Dissemination and Antibody Responses of Rhesus Macaques Exposed to the Human Gammaretrovirus XMRV<\/u><\/strong>\u00a0\u2013 This research from Emory University showed that murine leukemia viruses tend to disappear quickly from the blood, settling in specific organs, but can easily be reactivated by immune stimulation<\/strong>.\u00a0http:\/\/jvi.asm.org\/content\/early\/2011\/02\/16\/JVI.02411-10.abstract<\/a><\/b><\/p>\n 9.<\/strong>\u00a0\u00a0The Rise in Autism and the Role of Age at Diagnosis<\/u>\u00a0\u2013\u00a0This 2009 article from the journalEpidemiology<\/i>\u00a0\u00a0by Dr. Irva Hertz-Picciotto of the MIND Institute of the University of California, Davis tracked autism cases identified in California from 1990-2006. Picciotto and her team found that the rate of autism increased from a rate of approximately 1 in every 1,600 births in 1991 to a rate of approximately 1 in every 235 births in 2001 and the increase showed no signs of slowing.\u00a0 In April of 2013 the CDC reported that the estimated rate of autism is now\u00a01 in every 50\u00a0births. \u00a0In an article published in\u00a0Scientific American<\/em>\u00a0on January 9, 2009, Dr. Hertz-Picciotto noted that while several studies had suggested\u00a0the increase in autism was due to increasing chemical exposures, \u201cIn addition, fetuses and infants might be exposed to a fairly new infectious microbe, such as a virus or bacterium, that could be altering the immune system or brain structure.\u201d\u00a0\u00a0http:\/\/journals.lww.com\/epidem\/Abstract\/2009\/01000\/The_Rise_in_Autism_and_the_Role_of_Age_at.16.aspx<\/a><\/strong><\/p>\n 10<\/strong>.\u00a0\u00a0The Exotic Biology of Xenotropic Murine Leukemia Related Viruses: Pitfalls and New Concepts<\/u>\u00a0\u2013 This presentation from Dr. Judy A. Mikovits at the Mount Sinai Conference in November of 2013 details the very latest findings on the exotic biology of the murine leukemia retroviruses and the role they may play in many human diseases.\u00a0(44 slides):<\/strong>\u00a0\u00a0http:\/\/www.facebook.com\/media\/set\/?set=a.244564049033185.1073741854.101589063330685&type=1<\/a><\/strong><\/p>\n \u200b11. \u00a0<\/strong>Evaluation of the Broad-Range PCR-Electrospray Ionization Mass Spectrometry (PCR\/ESI-MS) System and Virus Microarrays for Virus Detection<\/u><\/strong>\u00a0\u2013 This research from the University of California, Berkeley, and the Food and Drug Administration (as well as a very significant assist from Dr. Crystal Jaing of the Lawrence-Livermore National Laboratories) illuminates the best practices for viral detection and some unsuspected dangers.<\/strong><\/p>\n \u200bFrom the conclusions section, \u201cIn addition to the LOD studies, we used the new virus detection methods to analyze some vaccine-related cell substrates for the presence of novel viruses. Total nucleic acids were used for the detection of both DNA and RNA viruses. Analysis of cell lines by PLEX-ID and virus microarrays indicated the presence of various retrovirus-related sequences. However, the origin of these sequences could be attributed to the presence of cellular DNA containing endogenous retroviral sequences or viral transcripts that may be expressed constitutively from some endogenous retroviral DNAs (proviruses). However, this situation is applicable to all nucleic acid-based assays that can detect retroviruses. To assess the origin and nature of the retroviral sequences a follow up strategy using PCR, sequencing, and bioinformatics was developed. The analysis revealed that the unexpected RD114 sequences detected by PLEX-ID in canine cell lines were associated with endogenous gammaretroviruses . . . Our analysis showed that different viruses were detected by using different technologies. These results emphasize the need to use a combination strategy for virus detection in evaluating the safety of biologicals or screening patient samples.\u201d<\/strong><\/p>\n This research strongly supports a combination strategy to detect novel viruses in biological products like the cell substrates used in vaccines as well as when investigating suspect disease populations who may be suffering from some type of viral infection. \u200b<\/strong>http:\/\/www.mdpi.com\/1999-4915\/6\/5\/1876\/htm<\/a><\/strong><\/p>\n 12. \u00a0How Can Xenotropic Murine Leukemia Virus-Related Virus (XMRV) Contamination be Prevented?<\/u>\u00a0\u2013 This article published in February of 2014 in the journal of the American Society of Microbiology addresses the question of how to prevent both laboratory contamination by XMRV as well as prevent the infection of lab workers. \u00a0The article describes XMRV as a lab creation which is capable of \u201cinfecting human cells and integrating its genome into that of the host. \u00a0Therefore, XMRV virions and XMRV-infected cells are considered as biosafety level 2 organisms.\u201d\u00a0\u00a0\u00a0http:\/\/aem.asm.org\/content\/early\/2014\/02\/10\/AEM.04064-13.abstract<\/a><\/strong><\/p>\n 13. \u00a0Origins of the Endogenous and Infectious Laboratory Mouse Gammaretroviruses<\/u>\u00a0\u2013\u00a0 Successful interspecies transmissions can produce disease in new hosts that are unprepared to resist unfamiliar and potentially pathogenic agents.\u00a0 Xenotropic MuLVs (X-MuLVs) were later isolated by Levy and Pincus from the NZB mouse strain [2], and were termed \u201cxenotropic\u201d because they could infect cells of multiple species, such as human, rabbit and cat, but were unable to infect cells of the mice from which they were isolated. While these early observations suggested that P-MuLVs have the broadest host range of the MuLV subgroups, more recent studies have shown that X-MuLVs can actually infect more mammalian species than P-MuLVs, and that X-MuLVs but not P-MuLVs are capable of infecting all wild mouse taxa [8] (Table 1).<\/strong>http:\/\/www.mdpi.com\/1999-4915\/7\/1<\/a><\/strong><\/p>\n![\"e331087f83bd6cc644b204c575680f8f\"](\"http:\/\/circleofdocs.com\/wp-content\/uploads\/2015\/12\/e331087f83bd6cc644b204c575680f8f.jpg\")
<\/a>\u00a0Dr Judy Mikovits<\/a>, PhD is a Biochemist, cellular and molecular biologist with over 30 years of scientific expertise. She has directed programs on HIV, cancer, epigenetics, and neuroimmune disease, with a focus on development of novel drug and diagnostic technologies. She is an award winning book called\u00a0Plague<\/a>.<\/p>\nhttp:\/\/www.sciencedirect.com\/science\/article\/pii\/S1931312809003540<\/a><\/strong><\/pre>\n<\/div>\n